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2005 PhD in Pharmacology, Goethe University Frankfurt
2005 Postdoctoral Fellow, Pharmacology, Goethe University Frankfurt and Department of Neurobiology, University of Alabama, Birmingham/USA
2011 Professor, FAU Erlangen-Nürnberg
2017 Professor, Pharmacology & Toxicology, Johannes Gutenberg University Mainz
Research in my group aims to understand the pathophysiology of neuropsychiatric diseases e.g. mood disorders and Alzheimer disease with a focus on ion channels and mitochondria. In addition, we aim to develop new pharmacological therapies for these diseases.
Interaction of ASM/ceramide system and TRPC6 channels and its role in plasticity and depression-like behavior
Department of Neurology
Chair of Neurology
1999 MD, University of Freiburg (Breisgau)
2007 Specialization in Neurology at the University Hospitals in Würzburg, Göttingen and Bochum
2008 Head of the Neuroimmunology Laboratory, Department of Neurology, FAU Erlangen-Nürnberg
2010 Head of the Neuroimmunology Section, Department of Neurology, FAU Erlangen-Nürnberg
2013 Professor for Neuroimmunology, FAU Erlangen-Nürnberg
2018 Chair of Neurology, University Hospital Regensburg
My continuing scientific interests include neurodegeneration and neuroprotection as well as glial cells in MS, and mechanisms of action of new immunotherapies.
Role of the transcription factors Sox8 and Sox10 in susceptibility and repair in demyelinating diseases
Department of Biology
Chair of Animal Physiology
- Phone number: +49 9131 8528329
- Email: email@example.com
- Website: http://www.tierphys.nat.uni-erlangen.de/staff/regus-leidig.shtml
2008 PhD, Max Planck Institute for Brain Research, Frankfurt/Main, and FAU Erlangen-Nürnberg
2008 University Assistant, Department of Biology, FAU Erlangen-Nürnberg
2014 Habilitation in Zoology, FAU Erlangen-Nürnberg
2014 Group Leader, Department of Biology, FAU Erlangen-Nürnberg
2018 HTA Writing Expert, Novartis, Nürnberg
Former research focus
The research of the Brandstätter and Regus-Leidig group focuses on molecules and mechanisms that play a role in the development and the structural and functional organization of chemical synapses in the CNS with a special focus on the retina. In our experimental approach, we combine methods ranging from immunocytochemistry and light- and electron microscopical imaging to biochemistry, cell and molecular biology, and electrophysiology.
As synaptophathies = malfunctioning synapses are a reason for many neurodegenerative diseases and neurological disorders, the aim of our research is to contribute to a better understanding of synapse function in health and disease.
Examining the synaptogenesis and synapse maintenance factor Bassoon as a putative factor for the differential sensitivity of photoreceptors to late-onset degeneration